When I asked myself the question, “How does neurogenesis work after trauma,” I ended up here, where I read:

Traumatic brain injury stimulates an increase in the proliferation of endogenous NSCs, particularly in the neocortex where it does not normally occur, suggesting that the adult brain has the inherent potential to restore populations lost to injury and that it may become possible to manipulate endogenous multipotent precursors in situ to replace lost or damaged neurons. Our data show that NSC-protein markers are found in the cells located in normal cortex, but these cells barely express proliferative markers such as Ki67, suggesting that, even if there are some NSCs in the normal cortex in humans, the majority are in quiescent.

These are the words I’d like to explore more in future posts, based upon my research:




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